Publications
Filters: First Letter Of Title is O [Clear All Filters]
Obesity and corticosteroids: 11beta-hydroxysteroid type 1 as a cause and therapeutic target in metabolic disease. Mol Cell Endocrinol. 2010;316(2):154-64. Abstract
Obesogens and obesity: An alternative view. Obesity. 2013.
Observing the NICEties of hypertension management. J R Coll Physicians Edinb. 2012;42(3):251-7. Abstract
Oestrogen and benign prostatic hyperplasia: effects on stromal cell proliferation and local formation from androgen. The Journal of endocrinology. 2008;197:483-91. Abstract
Oestrogen and the cardiovascular system: the good, the bad and the puzzling. Trends in pharmacological sciences. 2001;22:152-6. Abstract
Oestrogen-mediated cardioprotection following ischaemia and reperfusion is mimicked by an oestrogen receptor (ER)alpha agonist and unaffected by an ER beta antagonist. The Journal of endocrinology. 2008;197:493-501. Abstract
Omental 11beta-hydroxysteroid dehydrogenase 1 correlates with fat cell size independently of obesity. Obesity (Silver Spring). 2007;15(5):1155-63. Abstract
Ontogenetic regulation of mouse Ren-2d renin gene in transgenic hypertensive rats, TGR(mREN2)27. The American journal of physiology. 1993;265:E699-707. Abstract
Ontogenic emergence of a quail leukocyte/endothelium cell surface antigen. Cell Differ. 1988;23(3):165-74. Abstract
Ontogeny of 11beta-hydroxysteroid dehydrogenase: activity in the placenta, kidney, colon of fetal rats and rabbits. Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et métabolisme. 2001;33:78-83. Abstract
Ontogeny of the neurosteroid enzyme Cyp7b in the mouse. Molecular and cellular endocrinology. 2001;174:137-44. Abstract
Optimal antithrombotic treatment during primary percutaneous coronary intervention? Heart. 2011;97(18):1459-60.
Optimal antithrombotic treatment during primary percutaneous coronary intervention? Heart. 2011;97(18):1459-60.
Optimisation of growth hormone production by muscle cells using plasmid DNA. The Journal of endocrinology. 2000;165:329-36. Abstract
Osteoporosis is a major confounder in observational studies investigating bisphosphonate therapy in aortic stenosis. J Am Coll Cardiol. 2012;60(11):1027; author reply 1027.


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